PRION-LIKE DISEASES. “Five critical molecules have been studied in this regard—amyloid and tau in Alzheimer’s, alpha-synuclein in Parkinson’s, SOD1 in ALS, and polyglutamine in Huntington’s. Just like PrP each have normal functions in the brain and, then, when misfolded, become prions. It appears that amyloid beta, tau and alph-synuclein influence each other in Alzheimer’s and Parkinson’s.”
https://lnkd.in/dKV3Xez
linkedin post 2022-06-29 05:12:48
Nigel Fleming
September 21, 2022